Role of Forces Rather than Motion and Posture in Foot Orthotic

Transcription

Role of Forces Rather than Motion and Posture in Foot Orthotic
Role of Forces Rather than
Motion and Posture in Foot
Orthotic Prescribing
Craig Payne
Running
§  Principles applied elsewhere
§  Fascination with social media fanaticism –
voyeuristic!
§  Fallacies: cherry picking; confirmation biases;
appeal to authority; etc
§  Evidence vs anonymous bloggers
§  ‘Turmoil’ in the ‘industry’
§  Trends away from foot orthotics
Overuse Injuries
§  Affect 30-70% of runners in any one
year
Why:
§  Cumulative stress in the tissues
beyond what the tissues can tolerate
Factors:
running footwear used and issues with them
the training routine
overtraining
the particular running technique or form used by the
runner
§  muscle strength imbalances and flexibility issues;
§  issues with foot and lower limb biomechanics or alignment
§  issues with the proximal control of lower limb
biomechanics
§ 
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§ 
§ 
Traditionally:
§  Role of foot orthotics was to change
alignment or posture of the foot
Principle
What is the evidence that foot prona1on is even a problem? — Cross sec1onal studies: — Bunions and ‘flat feet’ (Kalen & Brechner; 1988; Inman, 1976; Goldner & Gaines, 1976) — MTSS (‘shin splints’) and pronated feet (ViLasalo and Kvist, 1983; Messier and PiLala, 1988)
— No correla1on (Rome et al, 2001; Hogan et al 2002) — Pronated foot protec1ve (Cain et al, 2006) What is the evidence that foot prona1on is even a problem? — Prospec1ve studies: — No rela1onship between foot prona1on and overuse injuries (Cowan et al, 1992; Cowan et al, 1996; Brusseuil et al, 1998; Wen et al, 1998; Twellaar et al, 1997; Kaufmann, Brodine & Shaffer, 1999; Michelson, Durant & McFarland, 2002; Giladi et al, 1985; Burns et al, 2005; Hetrsroni et al, 2006) — Weak rela1onship between foot prona1on and overuse injuries (White & Yates, 2002; Reinking 2006, Willems et al, 2007) Do foot orthoses change even change rearfoot motion?
§  They don’t change rearfoot kinematics (eg Rodgers &
Leveau, 1982; Blake & Ferguson, 1993; Brown et al,
1995; Nawoczenski et al, 1995; Nigg et al, 1997; Butler
et al, 2003; Stackhouse et al, 2003; Williams et al
2003…)
§  They change rearfoot kinematics (eg Bates et al, 1979;
Smith et al, 1986; Novick & Kelly, 1990; McCulloch et
al, 1993; Stell & Buckley, 1998; Leung et al, 1998;
Genova & Gross, 2000; Nester et al, 2001; Woodburn
et al, 2003…)
§  and when they do change rearfoot kinematics, its small
à biological significance?
Rearfoot mo1on changes and clinical outcomes •  Zammit GV & Payne CB: Rela1onship Between Posi1ve Clinical Outcomes of Foot Ortho1c Treatment and Changes in Rearfoot Kinema1cs. J Am Podiatr Med Assoc 2007 97: 207-­‐212 RCT’s; outcomes studies; patient satisfaction studies; eg
§  Blake & Denton (1985); Survey; Orthoses definitely
helped 70%; 78% felt that their devices improved their
posture
§  Donatelli et al (1988); 81 subjects; retrospective
survey; 91% satisfied with their foot orthoses; 94% still
wearing the foot orthoses; 52% would “not leave home
without them”
§  Mororas & Hodge (1993); prospective survey of 523;
83% satisfied with their orthoses; at 14 weeks post
issue 63% had their symptoms completely resolved and
95% completely or partially resolved.
§  Etc; etc; etc; etc; etc; etc; etc; etc; etc; etc; etc
Measuring force needed to supinate the foot
§  If alterations in the forces are important
clinically, how can that be applied?
§  Rather than focus on magnitude of
excessive foot pronation, may need to focus
on measuring the force needed to supinate
the foot (‘supination resistance’)
§  ‘Supination resistance’ is an example of one
of the forces that can be clinically estimated
(windlass forces are another).
Supina1on Resistance Test •  Supina)on resistance tes)ng: •  60-­‐350N range 60-350N
Unilateral pathology
(Payne et al, 2002)
§  Subjects – unilateral lower limb pathology that
could be to what is assumed as being due to
excessive pronation of the foot
§  n=28
§  FPI > on symptomatic side 15/24 (= in 4)
§  Supination resistance > on symptomatic side
25/28
§  p=0.012
§  Conclusion: Pronatory force more predictive of
symptomatic side that pronated position
Force needed to supinate the foot •  Posterior 1bial dysfunc1on group: –  328 (+21) Newtons (n=14) •  Reference group: –  138 (+46) Newtons (n=142) Peroneal tendonitis
Peroneal tendonitis
§  n=13
§  Mean FPI = 5.6 (+2.7)
§  Mean supination resistance 91 (+21)N
§  (reference population 138 (+46) N)
Conclusion:
§  Foot is pronated, but force needed to supinate the foot is
low (peroneals may have to work harder)
Implications:
§  May need to increase pronatory force on lateral side of
rearfoot (despite pronated position)
Peroneal tendonitis
Hypothesis?
§  The foot orthoses (~running shoe) need
design parameters that match the
supination resistance force
§  rigidity and/or inverted position of
orthotic
supination resistance
§  ‘motion control’ features in running
shoes
supination resistance
§  How test this? How to apply this
clinically?
Traditional Foot Orthotic Prescribing
§  Based on:
• decision making to change alignment or func1on of the foot • various clinical tests or observa1ons were used to derive the prescrip1on variables • design features were incorporated into the foot ortho1c to move the foot closer to what was theore1cally considered a normal or ideal structural alignment or func1on Pathology Specific Prescribing
§  Tissue stress model
§  based on the concept of the decision
making is based on the pathology present
and not necessarily on the structural
alignment or function of the foot.
§  Design features are used in the foot
orthoses to alter the forces through the
pathologic tissues to reduce the load to
below a level that the tissues can tolerate.
Summary
§  Clinical tests à prescription variables
à How best to deliver those prescription
variables (the design features)
Concept
§  Clinical test / Pathology Specific
Prescribing
§  Prescription variable
§  Design Feature
Orthotic Prescribing:
— Supination resistance (Clinical test)
— If high, need forces to match it (Prescription
Variable)
— Medial wedging designs; more rigid orthotics
(Design features)
Types of foot orthoses
— Arch cookies, medial wedges
— Triplane wedges
— LSR prefab’s (eg Formthotics, Vasyli)
— Direct molded devices
— FFO’s (Root & modified Root type
devices)
— HSR prefabs (eg Prothotic, Interpod)
— Kirby medial skive, MOSI, Blake inverted,
DC wedge
— (Surgical)
More control, force or aggressive
Unilateral plantar fascii1s and the force to establish the windlass •  n=12 •  Windlass tension: Degrees of
hallux
dorsiflexion
Asymptomatic
side (newtons)
Symptomatic
side (newtons)
0
4
8
12
20
30
4.6
10.4
19.7
34.7
56.5
75.1
7.9
19.2
37.5
59.3
86.0
119.8
Why is low-dye strapping so effective?
Orthotic Prescribing:
— Plantar fasciitis (Pathology specific prescribing)
— Lower the forces in the plantar
fascisa(Prescription Variable)
— Invert rearfoot; forefoot valgus post; 2-5 bar; met
dome; reverse Mortons; cuboid elevation; first ray
cut out (Design features)
Plantar fasciitis prescription
variables:
— Need to lower the forces through the
plantar fascia:
1.  Low dye strapping
2.  Invert rearfoot and evert the forefoot
Think Intuitively
§  Motion does not damage tissues
§  Foot posture/alignment does not
damage tissues
§  Forces/loads damage tissues
§  Kinetics hurt; kinematics doesn’t
Clinically
§  Clinical tests to determine the loadsà
are there prescription variables that
can be designed into a foot orthoses to
reduce that load?
Overuse Injuries
Why:
§  Cumulative load in the tissues beyond
what the tissues can tolerate
Role of Foot Orthotics:
§  Reduce that load
Turmoil in the running ‘industry’
§  Barefoot running (minimalism)
§  “Foot orthotics are evil and should be
banned”
§  “Foot orthotics weaken muscles”
§  “Running shoes weaken muscles”
§  Proximal control
§  Running ‘form’
§  Social media debates
What have we learnt
§  “Cumulative load in the tissues beyond
what the tissues can tolerate”
§  à increase the ability of the tissues to
take the load
§  à other strategies to reduce the load
§  à short vs long term use of foot
orthotics
Clinical tests to derive prescription
variables:
§  Supination resistance
§  Windlass mechanism forces
§  Lunge test
§  Navicular drop and drift
Delivery of the design features
§  There is no one best orthotic,
material, manufacturing process etc
§  The best one is the one that has the
design features that deliver the
variables that are needed for that
patient
§  Does it matter how you deliver them?
Design Features
§  Resist the pronatory forces (ie medial
wedging; material rigidity)
§  Facilitate the windlass (ie lateral
column elevation; Cluffy wedge)
§  etc
Specific Pathologies
§  Plantar fasciitis
§  Peroneal tendonitis
§  Medial tibial stress syndrome
§  Anterior compartment syndrome
§  Patellofemoral pain syndrome
§  Posterior tibial tendonitis
§  “Top of foot pain”
Conclusion
§  Think re forces in the tissues rather than foot
posture and alignment
§  Think re clinical tests to derive prescription
variables
§  Think re what design features are needed to
deliver those variables
§  Think re increase the ability of the tissue to take
the load
§  Think re other strategies to decrease the load
§  http://www.runresearchjunkie.com/
§  http://www.podiatry-arena.com
§ 
@CraigBPayne
§ 
/craig.payne.3958
§  [email protected]