Pediatric Orthopedics Potpourri - Healthcare Professionals
Transcription
Pediatric Orthopedics Potpourri - Healthcare Professionals
Pediatric Orthopaedics When to send… CHRIS SOUDER, MD BAYLOR SCOTT & WHITE HEALTH MCLANE CHILDREN’S HOSPITAL Orthopaedics Specialty originated in the diagnosis and treatment of conditions of children Ortho To straighten or correct Paed Child, children Orthopaedics Trauma Fractures Ligament & joint injuries Infections Osteomyelitis Septic Arthritis Spine Scoliosis Kyphosis Spondylolysis Foot & Ankle Clubfoot Congenital vertical talus Tarsal Coalitions Cavus & Pes planus (flatfoot) Hip Developmental dysplasia (DDH) SCFE Perthes Sports Medicine ACL, meniscus tears Patellofemoral instability Femoroacetabular impingement Upper Extremity Syndactyly, polydactyly Congenital deformities Neuromuscular Cerebral Palsy Spina bifida And more… Orthopaedics Trauma Fractures Ligament & joint injuries Infections Osteomyelitis Septic Arthritis Spine Scoliosis Kyphosis Spondylolysis Foot & Ankle Clubfoot Congenital vertical talus Tarsal Coalitions Cavus & Pes planus (flatfoot) Hip Developmental dysplasia (DDH) SCFE Perthes Sports Medicine ACL, meniscus tears Patellofemoral instability Femoroacetabular impingement Upper Extremity Syndactyly, polydactyly Congenital deformities Neuromuscular Cerebral Palsy Spina bifida And more… Most Common Things I See (and don’t operate on) Anterior knee pain In-toeing Knock-knees/Bowlegs Flatfeet Anterior Knee Pain Chondromalacia Patella Pain in the knee due to increased pressure from the patella Commonly occurs without injury to the knee Pain is diffuse about the knee I tend to see kids draw it out superiorly and lateral to the patella Described as achy May worsen with prolonged sitting or with activities May have catching or giving out episodes Minimal or no swelling present Anterior Knee Pain Physical Exam Pain with compression or manipulation of the patella Diffuse TTP No significant effusion NO pain with ROM of hip Tight hamstrings and quads Decreased popliteal angles (straight leg raise) and prone knee flexion Treatment NSAIDs I typically ask them to take these as a scheduled Rx for 2-3 wks Ice/heat Stretching & Strengthening Anterior Knee Pain Hamstring Stretches Quadricep Stretches In-toeing Metatarsus Adductus Internal Tibial Torsion Femoral Anteversion In-toeing Metatarsus Adductus Most common in infants/toddlers Typically resolves spontaneously Stretching may or may not help Rarely are corrective shoes required Surgery performed only in severe and persistent cases Requires osteotomies Child typically > 4 y/o In-toeing Internal Tibial Torsion Inward twist of the tibia Most common cause in 3-4 y/o Most cases slowly improve over time without treatment Infants are born with medially rotated feet that laterally rotate as they age No treatment needed Identified by a decreased thigh foot axis (TFA) or decreased transmalleolar axis In-toeing Femoral Anteversion Infants are born with femoral anteversion of ~40° that typically decreases to ~15° by adulthood Lower extremity internally rotates to compensate for increase anteversion Patella rotated inwards and in-toeing present in stance phase of gait Commonly seen in children 5-6 y/o Typically improves by 10 y/o Recommend improve sitting Surgery rarely needed for symptomatic patients 11-12 y/o Requires femoral osteotomy Knock-knees / Bowlegs Knock-knees Genu valgum Bowlegs Genu Varum Children naturally progress from varus to valgus during development Concern arises if: Significant varus past 2 y/o Unilateral Severe Familial Flatfeet Pes Planovalgus One of the most common “deformities” evaluated by pediatric orthopaedists Normal variation of childhood Normal in children as arch slowly develops over time Frequently associated with ligamentous laxity Deformity corrects with heel rise Feet that do not correct need to be seen by an orthopaedic surgeon Treatment is conservative Studies have proven no benefit with corrective shoes or arch supports Symptomatic tx can consist of well-structured athletic shoes or off-the-shelf arch supports Surgery needed only in severe cases (>8 y/o) Trauma 25% of children sustain a injury every year 10-25% of injuries are fractures Largest share of all children injuries Radius is most common long bone fracture 23% of all fractures 21.7% of fractures involve the physis Trauma Most children factures can be treated with nonoperative methods Trends toward more operative treatment Improvement in technology Rapid healing allows for minimal internal fixation Modern parents expect the perfect outcome Trauma Unique aspects Anatomical regions of a growing bone Different fracture patterns Physeal fractures Remodeling Children Fractures Aids in communication & description of fx Classifications Guides treatment Acceptable criteria Length of immobilizatoin Provides prognosis Potential for remodeling Possible complications Children Fractures Plastic deformation Greenstick fracture Buckle (Torus) fracture Physeal Fractures 20-30% of all children fractures Salter-Harris Classification Guides treatment Provides prognosis Reduction is primarily traction Physeal disturbance Harris growth arrest lines Need re-evaluated at 6 & 12 mc Remodeling Bone and cartilage remodel in response to normal stresses of body Body weight Muscle action Joint reactive forces Greater potential Younger Closer to physis In plane of joint motion Rotational deformities do not reliably remodel Common Injuries Distal Radial Buckle Fracture Supracondylar Humerus Fracture Unique Injuries Occult elbow fracture Posterior fat pad sign with history of trauma to the elbow No radiographic evidence of fx Tx is long arm splint or cast for 3 weeks 76% have evidence of fx 53% SCH 26% proximal ulna 12% lateral condyle 9% radial neck Unique Injuries Radiocapitellar dislocation Monteggia Fracture-dislocation Unique Injuries Tillaux Fracture Triplane Fracture Key Points Fractures near physis heal rapidly Remodeling potential allows for ‘acceptable deformity’ Rare stiffness seen with immobilization Physeal arrest can lead to deformity Must remember the possibility of child abuse Compartment Syndrome 3 As Increased Anxiety Increased Analgesic requirements Increased Agitiation Acute Hematogenous Osteomyelitis (AHO) Most commonly occurs in the metaphysis 75% of cases involve long bones Lower extremities more frequent than upper Most common pathogen is Staph Aureus Commonly seen after minor trauma Typically present with pain and decreased use of extremity Fever is commonly seen Skin changes can be present Acute Hematogenous Osteomyelitis (AHO) Labs WBC ESR Least sensitive Becomes elevated after 48h CRP Serum levels rise after 6h Elevated in 98% of osteomyelitis Blood cultures Yield organisms 30-60% Acute Hematogenous Osteomyelitis (AHO) Imaging Radiographs Soft-tissue swelling & loss of tissue planes seen within 3 days Evaluate for associated effusion Bony changes present after 7-10days MRI Sensitivity 88-100% Specificity 75-100% IV contrast Allows evaluation for soft-tissue and subperiosteal abscess formation Acute Hematogenous Osteomyelitis (AHO) Treatment PO abx IV abx Needle aspiration Open biopsy Curettage Septic Arthritis Most commonly a result hematogenous seeding of the synovium Staph Aureus is most common pathogen Child is typically more ill appearing than in osteomyelitis Pain experienced with gentle ROM Same work up as for osteomyelitis Labs (ESR, CRP, WBC, BCx) Plain radiographs If concern for septic hip, obtain ultrasound to evaluate for an effusion Aspiration provides final diagnosis Treatment is URGENT joint lavage Transient Synovitis of the Hip One of the most common causes of hip pain Must be differentiated from septic arthritis Accurate diagnosis required to avoid joint destruction Clinical Predictors Refusal to bear weight Temp > 38.5°C WBC > 12,000 ESR > 40 mm/h CRP > 20.0 mg/L Scoliosis Curvature of the spine > 10° in the coronal plane Idiopathic Infantile <4 y/o Juvenile 4-10 y/o Adolescent 11 y/o & older Congenital Secondary to a bony abnormality Neuromuscular CP, syndromic, spina bifida, etc Adolescent Idiopathic Scoliosis (AIS) Larger curves requiring treatment are much more common in females Family history is associated with presence, not progression, of a curve Strongest relationship is a daughter of male with scoliosis Etiology is not completely understood AIS Curve progression associated with curve magnitude and growth remaining Seen during rapid growth phases Predicted based on Skeletal age • Risser sign, bone age Maturity • Onset of menses Indicates deceleration of growth AIS Screening Recommended to be performed 2x for females 10-12 y/o 1x for males 13-14 y/o Appearance Shoulder asymmetry Scapular prominence Waist asymmetry Scoliometer Rib or lumbar prominence on Adam Forward Bend Test 7° angle of trunk rotation (ATR) referred to Orthopaedics AIS Treatment Observation <25° with significant growth remaining (Risser 0-2) <45-50° with minimal growth remaining (Risser 3-5) Bracing >25° with significant growth remaining (Risser 0-2) Surgery >45-50° Most commonly posterior spinal instrumentation and fusion Key Points Goals of treatment Limit magnitude of curve/deformity present at skeletal maturity Curves <45-50° at maturity have a low likelihood of progression in adulthood Bracing is used to halt or slow progression Does not improve curve Patient expectations Braces are worn 12-23h/day for avg 12-18m Some deformity remains after surgical correction Patient Education http://www.settingscoliosisstraight.org/ Spondylolysis Spondylolysis Most commonly a result of a pars defect Tends to occur in adolescent athletes Stress fracture of the pars inter-articularis Associated with repetitive hyperextension Gymnasts, down linemen Produces dull low back pain Can radiate to buttocks or posterior thighs Worse with back extension Rare radicular complaints Often associated with hamstring tightness Spondylolisthesis Slippage of the vertebra secondary to spondylolysis Spondylolysis Mechanical back pain warrants radiographs Especially in patients at risk (gymnast, etc) Spot lateral of lumbosacral junction Treatment Avoid painful activities NSAIDs Ice/heat Stretches/strengthening Bracing Strengthen core and abdominal musculature If failed PT Surgery Last resort after months of conservative treatment Clubfoot Congenital talipes equinocavovarus One of the most common birth defects 1 in 400 babies in US Males 2x more frequent than females Can be unilateral or bilateral Etiology unknown Occasionally associated with other orthopaedic conditions DDH Clubfoot Congenital deformity of the foot consisting of Cavus of the midfoot Adductus of the of the forefoot (metatarsus adductus) Varus of the hindfoot Equinus of the hindfoot C-A-V-E Foot is twisted downward and inward Severity ranges from mild to severe Requires vigorous orthopaedic treatment Clubfoot Gold standard in treatment is corrective casting described by Dr. Ignacio Ponseti Allows soft-tissue relaxation and atraumatic remodeling of joint surfaces The earlier the treatment begun, higher likelihood of success <1 month of age Surgical correction rarely required Severe deformities and neglected feet <5% Associated with poorer outcomes Scarring, stiffness Ponseti Casting Consists of serial manipulations of the clubfoot to stretch the contracted tissues Casts are then applied to hold the correction Cast changes and manipulations occur weekly until foot is corrected Occasional Achilles tenotomy is needed to obtain correction of equinus Day surgery vs procedure room Clubfoot Bracing is required after casting for successful treatment Prevent recurrence Allow for remodeling of joints in corrected position Worn full time for several months Worn for nighttime and naps until 3-4 y/o Importance of brace cannot be overemphasized Poor compliance is #1 cause of recurrence Clubfoot Approximately 1/3 have partial recurrence Repeat casting can be performed if young Limited surgical correction can be performed 30% display a dynamic swing phase supination deformity as a young child Tendon transfer can prevent further deformity Clubfoot Tarsal Coalitions Abnormal connection between bones of the feet Most commonly between the calcaneus and navicular 2nd most common is talocalcaneal (subtalar) Usually become symptomatic in adolescence when the region begins to ossify and stiffen Pain is most common complaint Exacerbated with activities Worse with running on uneven surfaces Can be associated with recurrent ankle sprains Patient may note decrease ROM or deformity Flatfoot deformity Tarsal Coalition Physical Exam Rigid, uncorrectable flatfoot deformity Decreased subtalar ROM and ankle plantarflexion Peroneal spasms 50-60% have bilateral coalitions Radiographs can usually provide diagnosis AP, lateral, oblique, Harris view MRI or CT used if suspicion high and XRAYs neg Tarsal Coalition Initial trial of conservative treatment indicated for symptomatic coalitions Limit subtalar ROM for 4-6wks Short leg cast UCBL orthotic Surgery indicated for refractory cases Excision of coalition with interposition of muscle or fat Fusion occasionally needed for large coalitions Flatfeet Discussed earlier Developmental Dysplasia of the Hip (DDH) Formerly known as Congenital Dislocation of the Hip Spectrum of structural abnormalities about the hip presenting from birth to skeletal maturity Commonly due to excessive laxity of the hip with resultant instability of the femoral head within the acetabulum Leads to inadequate acetabular and/or proximal femoral development Typically begins with normal structures during embryogenesis Abnormalities then evolve overtime Infancy—subluxatable, dislocatable, dislocated Childhood—dysplasia, dislocated hip Adolescent—dysplasia DDH Incidence ranges from 1:1,000 to 3.4:100 live births Dislocation 1.4:1,000 Clinical findings 2.3:100 Ultrasound abnormality 8:100 Risk factors Family history Breech position (Feet first) 17-23% of DDH patients were breech Female gender Positive family history in 12-33% of cases 80% of cases are females First-born child Oligohydraminos Limited fetal movement Association with other intrauterine molding abnormalities • Torticollis and metatarsus adductus • Crowding phenomenon DDH Physical Exam Early Findings Ortolani sign palpable sensation of the femoral head reducing into the acetabulum • Trochanter is elevated as hip is abducted • Palpable clunk compared to audible click • Originally described as the femoral head gliding in and out of the acetabulum over the neolimbus Barlow sign palpable sensation of the femoral head dislocating from the acetabulum • Provocative maneuver with the femur adducted and flexed Newborn screenings suggest 1:100 newborns have some degree of instability (positive Ortolani or Barlow) DDH Physical Exam Late Findings Instability is absent due to adaptive changes Limited abduction is the most common, reliable finding Limb length discrepancy More specific than thigh folds Klisic sign Galeazzi sign reveals apparent femoral shortening Asymmetric gluteal folds Adductor longus becomes contracted secondary to hip subluxation/dislocation Difficult to detect if bilateral dislocations are present A line from GT to ASIS passes inferior to the umbilicus Waddling gait or hyperlordosis in ambulatory patients Courtesy of D. Wenger DDH Imaging Ultrasound used until ~6 m/o (until femoral head ossifies) X-RAYs utilized after 6 m/o Currently ultrasound screening of all infants for DDH is not being performed Expensive False positives Ultrasounds are performed on children with abnormalities on physical exam or at increase risk based on risk factors Dysplasia can still occur despite normal initial ultrasound DDH Goal of tx is to center the femoral head in the acetabulum to allow appropriate acetabular development Pavlik harness Typically used in 0-6 m/o Worn for 2-4 months 95% success if began in 1st month, 85% after 1st month Abduction orthosis Useful in dysplasia or subluxtion in 6-24m Operative tx used if >18-24 m/o or if bracing fails Closed reduction and casting Open reduction + pelvic and/or femoral osteotomies *20% residual dysplasia even with successful childhood tx Evaluation of the influence of straight-leg swaddling on development of the hip 112 rats divided into a control group and 3 “swaddled” groups Early swaddling (1st 5 days of life) Late swaddling (2nd 5 days of life) Prolonged swaddling (1st 10 days of life) Swaddling No cases of dysplasia identified in the control group Prolonged swaddling led to the most detrimental outcomes 36 of 44 with dysplasia or dislocation (82%) Early swaddling group revealed 21 of 44 hips with dysplasia (48%) Swaddling Increased severity of pathology associated with increased swaddling time and earlier initiation of swaddling Prolonged swaddling: 33 dislocations, 3 subluxations Early swaddling: 14 dislocations, 7 subluxations Late swaddling: 1 dislocation, 8 subluxations Swaddling Gross examination revealed a deformed acetabular cartilage complex in the prolonged swaddling group The iliac limb of the triradiate cartilage was more vertical than control group Acetabular wall slope and acetabular index increased Increased appositional cartilage growth within the acetabulum prevented a congruent hip joint All findings are similar to those seen in human DDH DDH International Hip Dysplasia Institute www.hipdysplasia.org It is the recommendation of the IHDI that infant hips should be positioned in slight flexion and abduction during swaddling DDH Slipped Capital Femoral Epiphysis (SCFE) Displacement of the proximal femoral epiphysis secondary to weakness at the physis Typically occurs during rapid growth Overweight, adolescent Boys 13-15 y/o (avg 14), girls 11-13y/o (avg 12) Males >> Females; L > R Bilateral involvement 20-25% ½ seen at initial presentation Stable SCFEs present with painless/mild limp, externally rotated foot, decreased ROM Need referal to ortho for surgical stabilization *BEWARE OF KNEE PAIN in overweight adolescent male Unstable SCFEs present with severe pain, unable to bear weight Needs URGENT referral to orthopaedics Legg-Calve-Perthes Disease (LCPD) Due to an unexplained, temporary loss of blood flow to the femoral head Typically occurs in male children ages 3-10 y/o Patient presents with a limp Can complain of pain and/or dec ROM CAN PRESENT AS KNEE PAIN Exacerbated by activity, relieved by rest Treatment ranges from PT to braces and casting to aggressive surgical tx with osteotomies Based on age of child and degree of involvement Anterior Cruciate Ligament Tears Recent dramatic increase in incidence Increased athletic involvement Increased recognition of the condition Frequently seen in noncontact activity Twisting injury “Heard/felt a pop” Typically associated with immediate, large effusion PE reveals increased translation with Lachmans test Non-op tx associated with further intra-articular injury and accelerated degeneration Surgical reconstruction recommend if patient desires to return to activities Open physes require special thought/consideration Meniscal Tears Isolated tears are not commonly seen Occur in 50% of ACL injuries Discoid meniscus are prone to tearing Presents with complaint of a loud snapping sensation PE reveals joint line TTP + effusion MRI confirms diagnosis Tx is surgical repair in most cases Patellofemoral Instability Typically occurs without injury 2:1 Female:Male ratio Can be secondary to quadricep weakness and/or bony deformity Genu valgum Ligamentous laxity 2% of population has trochlear dysplasia 85% of instability patients have trochlear dysplasia Non-op tx for first time dislocators Immobilized x 3wks, patellar sleeve & PT x 4-6wks MRI if persistent effusion for 3 weeks 50% chance of redislocation with non-op tx Discuss surgery if repeat dislocations occur Femoroacetabular impingement Abnormal bony contact between the proximal femur and acetabulum Slow onset intermittent groin pain that may occur after minor trauma Pain is exacerbated by athletic activity and prolonged walking. May be associated with pain with prolonged sitting or driving PE reveals positive ‘impingement sign’ Hip flexion, internal rotation, adduction Non-op Tx: activity modification, NSAIDs, PT can be counterproductive Surgery can consistent of arthroscopic vs open dislocation if pt fails non-op Thank you