Thanks For Stopping By - Friends of Parkinson`s
Transcription
Thanks For Stopping By - Friends of Parkinson`s
P The October 2015 Friends of Parkinson’s Volume 2 Issue 10 arkinson Post Living Well with Parkinson’s ~ An Integrative Approach Parkinson's Alpha-Synuclein Explained Thanks For Stopping By Across The Pond "All Shook Up" A Caregiver's Christmas Low-Level Pesticides Linked to Parkinson's Disease Friends of Parkinson’s • October 2015 • Page 1 The Parkinson Post Message From FRIENDS OF PARKINSON’S Inside this issue Subscribe and/or Donate Today (page 8) Thanks For Stopping By Hi there. Thanks for stopping by. I’m here to tell you a story about how I’ve overcome the obstacles that Parkinson’s disease presents daily. It’s a tale of loss and love, a story of strength and commitment in the face of what simply is an “incurable progressive neurological disorder.” Let that reality sink in for a second before you continue, please. See, Parkinson’s disease is different for everyone. I presented with “central pain” stiffness and rigidity while others get the shakes, or tremors. Over the last four years of having active PD., I’ve found that if I exercise on a regular basis and eat a proper diet that it is possible to greatly improve the quality of my life and of those around me. One of the greatest gifts I can give myself and those on this journey with me are to embrace life. To not let Parkinson’s disease define me but to let it be the fuel that helps me power through each day with the hope and dream that someday we will find a cure for PD and all disease that robs us of our ability to live life to the fullest. Parkinson’s disease cost me everything, my career, my family and home; however, it gave me the passion and insight to live the life I was meant to and to be strong enough to send this message. “Be brave, be bold and thrive in the life you have.” My Power, through commitment, goes out to my son and my Katelyn. I may have Parkinson’s disease and it doesn’t have me. Publisher Friends of Parkinson’s Staff Writer Lisa Vanderburg Executive Director Jamillah Ali-Rahman Contributing Writers Benjamin Prewitt D. Joseph Mercola Advertising 725-222-8181 Low-Level Pesticides Linked To Parkinson's Disease By Dr. Joseph Mercola Page 3 Parkinson's… Alpha-Synuclein Explained By Lisa Vanderburg Pages 4 & 5 across the pond with Lisa Vanderburg Page 7 Benjamin Prewitt is an established artist. Diagnosed with Parkinson’s November 2010 at age 40, his approach is gentle, retrospective and always filled with beauty. A part of Benjamin Prewitt’s artist’s statement tells us much about the artist when he states, “Art” reminds us the seemingly opposite forces of joy and sorrow work in symmetry to inspire us to find balance in who we are. My greatest sorrow, Parkinson’s disease, does not define me; I am simply an artist with Parkinson’s. The two are not mutually inclusive, they just are. Benjamin’s blog and art “A life in progress” Post in responds to the Power through Parkinson’s blog soon to be syndicated and published through the BGF Foundation. www.BenjaminPrewitt.com. Distribution Manager Jon Carl Olson Page 2 Events & Resources Benjamin 2015 Int’l Research Specialist Lisa Vanderburg By Benjamin Prewitt Page 6 Much love and light. Editor Michelene K. Bell Message From Friends of Parkinson's Thanks For Stopping By Featured story Alpha-Synuclein Explained By Lisa Vanderburg Advisory Board Donna Jensen Dir. of Operations Frank Fuentes Dir. Marketing & Pub. Relations Lanie Spradlin Dir. Community Affairs Office The Parkinson Post 2400 N. Tenaya Way Las Vegas, NV 89128 Ph: 725-222-8181 Fx: 702-838-2640 Jim DiFiore Dir. Business Development E-mail [email protected] Charles Jensen Legal Advisor Website FriendsofParkinsons.org Shirley Hofmeister Dir. Emeritus Tina Drago Dir. Medical Services Page 2 • October 2015 • Friends of Parkinson’s Alpha-Synuclein is a sticky protein found throughout the body, but the mother lode is in the brain, where, if all goes smoothly, it's part of a process that leads to the release of dopamine… Don’t forget the Sock Hop! Save this Date: October 24, 2015 The Parkinson Post Low-Level Pesticides Linked to Parkinson's Dr. Joseph Mercola The risk of Parkinson’s disease clearly increases with exposure to certain environmental toxins, such as “Epidemiological research has identified several factors that may be linked to Parkinson’s, including rural living, well water, manganese and pesticides." pesticides. These pesticides, herbicides, and fungicides are potent toxicants that may cause disruptions or damage the neurological system, including your brain. What is perhaps most concerning is ambient exposure to pesticides has been found to increase the risk of Parkinson’s considerably; which was further confirmed by new research linking the disease to extremely low-level pesticide exposure. Eleven Commonly Used Pesticides Linked to Parkinson’s, Even at Low Levels Last year, UCLA researchers linked a fungicide known as benomyl to PD [Parkinson’s disease]. The US Environmental Protection Agency (EPA) banned the chemical after it was determined to be a potential carcinogen. That study found that benomyl blocks the aldehyde dehydrogenase (ALDH) enzyme. When working properly, ALDH changes aldehydes, which are toxic, to dopamine cells, into less toxic compounds. When ALDH is blocked, however, this transformation does not occur, contributing to the development of Parkinson’s. For the most recent study, researchers revealed eleven pesticides in all that inhibit ALDH and increase the risk of Parkinson’s disease. The effects were seen even with very low-level exposure – levels lower than are currently being used in everyday applications. The study’s lead author said: “We were very surprised that so many pesticides inhibited ALDH and at quite low concentrations that were way below what was needed for the pesticides to do their job… These pesticides are ubiquitous, and can be found on our food supply and are used in parks and golf courses and in pest control inside buildings and homes. This significantly broadens the number of people at risk.” Common Genetic Variant May Raise Parkinson’s Risk Six-Fold Certain people appear to be especially sensitive to the effects ALDH-blocking effects of pesticides. The study revealed people with a common genetic variant of the ALDH2 gene had a two to six times greater risk of developing Parkinson’s disease when exposed to pesticides compared to those without the genetic variant. The study reported: “ALDH inhibition appears to be an important mechanism by which these environmental toxins contribute to Parkinson’s pathogenesis, especially in genetically vulnerable individuals.” Unfortunately, unless you’ve been specifically tested, you’ll have no way of knowing whether you have this particular genetic variant. Moreover, even those who do have it will have a hard time completely avoiding pesticide exposure, since their use is ubiquitous. Can We Still Say Parkinson’s Disease Has No Identifiable Cause? Parkinson’s disease is still classified as idiopathic, meaning it has no identifiable cause. However, one reason it is likely on the rise is due to many environmental toxins that now bombard your body on a daily basis, with pesticide exposure becoming an undeniable risk factor. According to the Parkinson’s Disease Foundation: “Epidemiological research has identified several factors that may be linked to Parkinson’s, including rural living, well water, manganese and pesticides. Some studies have demonstrated that prolonged occupational exposure to certain chemicals is associated with an elevated risk of PD. These include the insecticides permethrin and beta-hexachlorocyclohexane (beta-HCH), the herbicides paraquat and 2,4-dichlorophenoxyacetic acids and the fungicide maneb. In 2009, the US Department of Veterans Affairs added Parkinson’s to a list of diseases possibly associated with exposure to Agent Orange.” Separate research has further revealed ambient exposure to organophosphate pesticides also increased the risk of developing Parkinson’s. Rotenone and paraquat are two additional pesticides linked to an increased risk of Parkinson’s disease, and both are lipophilic, meaning they resist breaking down in water and accumulate in your fat. Both are known to cross your blood-brain barrier. Dousing our crops with large amounts of glyphosate, the active ingredient in Monsanto’s broad-spectrum herbicide Roundup, may be another factor in the rising rates of Parkinson’s disease. These residues are found in virtually all food containing genetically modified ingredients. Avoiding pesticide exposure around your home, in your community, and via the food you eat, is clearly important for reducing your Parkinson’s risk, as is reducing your exposure to environmental toxins. Exposure to industrial solvents, including TCE, a common degreasing agent and dry-cleaning chemical, has also been linked to Parkinson’s disease for instance. Therefore, along with avoiding the use of solvents and pesticides in your home and garden, eating organic foods as much as possible will help you avoid unnecessary exposure to these chemicals. Another important and often-overlooked environmental risk factor is “silver” amalgam dental fillings, which actually are more than half mercury. Mercury becomes a biochemical train wreck in your body, causing your cell membranes to leak, and inhibits key enzymes your body needs for energy production and removal of toxins. Mercury toxicity can lead to major inflammation and chronic illnesses such as PD. Healthful Lifestyle May Lower Parkinson’s Risk Parkinson’s is a neurological disorder in which neurons in a region of dopaminecontinued on page 6 Friends of Parkinson’s • October 2015 • Page 3 The Parkinson Post Parkinson’s by Lisa Vanderburg Alpha-Synuclein Explained K eep it simple, stupid!’...that’s my motto. Don’t get me wrong – not for you, but for ME, because it takes ages to understand, much less absorb or translate! Being two stories short of a high-rise presents serious challenges and lots of ‘duh’ moments, but I think I got it? is aggregation - crap everywhere that gum-up the works. These aggregates are commonly known as Lewy bodies. Lewy bodies are present in a variant of Alzheimer's disease also known as Lewy body dementia, another disease known as multiple system atrophy or ‘neurodegenerative’, and, of course, Parkinson’s disease. So for all you peeps out there that wanna know the hoo-ha about alpha-synuclein…here it is. Now, that’s the simple version….without reference to other players like Tau and A-Beta proteins, or DNA strands, mitochondria, enzymes, or the many genes already identified as biomarkers for the disease. This will have to do to discuss the major issue, which is: Let’s gets a couple of abbreviations out of the way first: a-SYN = Alpha-Synucein PD = Parkinson’s ‘Aggregates’ are basically debris or detritus; organic matter produced by the decomposition of organisms. Alpha-Synuclein is a sticky protein found throughout the body, but the mother lode is in the brain, where, if it all goes smoothly, it’s part of a process that leads to the release of dopamine – that wonderful stuff that makes us feel good, controls movement and autonomic systems, all with the agility of a synchronized swimmer. Ideally, the a-SYN protein appears to function in the loading of vesicles (see graphic below) - small spherical bits of membrane that carry neurotransmitter molecules - to the pre-synaptic end of a neuron, which bumps Presynaptic into the business end Membrane of another neuron, synaptic allowing for the neurovesicle transmitters in the synaptic cleft vesicles to be released neurotransmitter into the synapse. Postsynaptic Membrane The top is the presynaptic neuron; the bottom, postsynaptic. They are both separate neurons in this chain gang. Okay. So what’s the big dealeo? Glad you asked. In Parkinson’s and specific other neurodegenerative diseases, it’s been found that a-SYN proteins are ‘misfolding’, creating clumps or aggregates. Like almost all proteins, after a-SYN is synthesized by the cellular machinery (i.e. ribosomes – they build long chains of amino acids, one at a time – exhausting!), it is supposed to fold in a certain way; the entire function of a-SYN is dependent on proper folding. So, if the folding goes wrong, then the protein fails to function – simple! The fall-out of this wayward behaviour Page 4 • October 2015 • Friends of Parkinson’s So it’s rather like loading a musket rifle or cannon; exact amount of powder shove a lead ball down the barrel specific grade of powder into the pan before closing the frizzen = success! Do it wrong = ka-boom… or maybe no head… Nah…bit sorta, er…permanent? Worse, these deviant, delinquent aggregates are not the only bits of junk that scupper any healthy a-SYN from assisting with neurotransmitter release from neurons. There’s the possibility of too much a-syn being produced as well, and that excess of a-SYN would lead to toxicity. Either way, its accumulation is a big problem in 90–95% of Parkinson’s patients. The other 5% are those that have direct-inherited disease (genetically passed down from parent to child) and for them, a-SYN is not considered a big player. Oh…did I mention, this toxicity is specific to neurons that release the neurotransmitter dopamine, and the aggregation may even be protective in other types of neurons? Okay, that makes no sense. I think what I’m saying here is that, on a cellular and molecular level, waste product is normal and dealt with in an orderly fashion. Not so with these a-SYN, simply because there’s too much junk. And another thing we have to throw into the mix; oxygenation. Say hello to ‘free-radicals’, which is what the dopamine neuron-specific toxicity appears to involve: the formation of The Parkinson Post Alpha-Synuclein is a sticky protein found throughout the body, but the mother lode is in the brain, where, if it all goes smoothly, it’s part of a process that leads to the release of dopamine… reactive oxygen, which can evolve during the breakdown of excess dopamine that builds up when it cannot be released from the neuron. Pharmaceutical companies like Neuropore Therapies, are teaming up with Belgian drug maker UCB, and QR Pharma, Inc. has something in the pipeline too. Okay, okay, but at a cellular level, it’s poison. It’s caustic and can cause the iron in our cells to basically go rusty. Yuck. Here’s where I may have to kneel on grits (ol’ Southern hospitality!): A similarly unexpected finding; one that’s got everybody hot and bothered, generating a great deal of interest, is that misfolded a-SYN are also found in our gut and olfactory (that’s ‘nose’ to you and me) system, which means they travel. To show you the significance of that, Patrik Brundin, MD, in 1988 received his PhD on intracerebral transplantation in PD. By 1992, he was part of a team that started neural transplants in PD patients. That worked well…for a time. But, and here’s the important bit, in 2008, they found Lewy bodies (these misfolded a-synuclein clusters) in the significantly younger (chronologically) grafts. Well, that opened up a whole NEW can o’ worms. Could it be this was Prion disease – neurodegenerative disorders causing inflammation and infection? Or maybe sub-structural faulty amino-acids could be a factor in a-SYN clumping? The body naturally deals with waste products – even on a cellular level, but these aggregates are too numerous. More importantly, it meant that, at some point during their travels, they were passing on a mutation to their buddies….and in turn suggested at some stage in this process, they were vulnerable. It was then a completely reasonable idea to think that bacteria might be involved. What kills bacteria? Antibodies, for one. That has led to some remarkable potential offerings in the Pharmaceutical field like NeuroPhage Technologies’ drug candidate NPT001 (bacteriophage M13), which are in the process of filing with the FDA; the first human trials expected in 2016. But, it requires administration to the brain directly, as crossing the blood-brain barrier (BBB) is a SOB. Most antibodies are too large to get in. Another offering is ICBI Technologies, and their SMART molecule (Specific Molecular Architecture for Recognition and Therapy Molecule), which has proven demonstration of BBB uptake in mice. Parkinson’s Resource Organization has been working closely with this Pharma since 2012. They keep us posted by way of ‘the Road to the Cure’ http://www.parkinsonsresource.org/featured-posts/ august-2015-update-on-the-road-to-the-cure/. That will be the latest, but they keep a running update from back to 2012. All the above have, at some stage or another, had significant grants from people like the MJFox Foundation. And I shoved poor MJ’s mug on three of my posts. Why? Because I wanted all the Orgs to work together under the umbrella of, say the World Parkinson’s Coalition. Interestingly, it was Jo Rosen, Founder & President of Parkinson’s Resource Organization, CA who schooled me on why all the PD groups, Assocs., Foundations & Institutes do NOT work well together…has a lot to do with agendas. Bleedin’ politicking!! Apologies, Michael J Fox! What did you ever do to attract my beady eye? Well…you got Parkinson’s for a start, and then went on to establish the biggest PD non-profit globally. And earnestly, those posts were for awareness, not target-practise. I think MJ himself would see that! But Jo Rosen – one feisty and savvy warrior told me “If ICBI had $5M today, it could be at human clinical trials in 18 to 24 months. I’ve already spent 3.5 years on this, Lisa. Imagine, had people opened their minds and pocket books 3.5 years ago, we would have been where we are still looking to be...’’ Now $5 million is a drop in the ocean in the scheme of things; chump-change for some. Anyway, after 30 years of basically the same old drug – Levadopa, somebody takin’ out the trash might well save us! With sincere thanks to Dr. Anuj Mankad, Molecular and Medical Geneticist, who so patiently picked up after I’d thrown all my toys outta the pram – thanks Doc, for the schooling! Lisa describes herself as a ‘chaotic and vaguely confused pit-bull’ who would ‘cheerfully throttle Parkinson’s Disease’ if she could! She is a European writer and the carer [caregiver] for her husband Pete, who she sees as a ‘shameless, wanton optimist’ – she knows what a blessing that is! She has now joined us as the International Research Manager and Staff Writer for Friends of Parkinson’s. Friends of Parkinson’s • October 2015 • Page 5 The Parkinson Post Lisa Vanderburg International Research Specialist for Friends of Parkinson's Across Pond the Sublime and Redonkulous: Parkinson's Well hello, fellow oddballs. I say that because PwP (people with Parkinson’s) and PwP/SD (that’ll be their caregivers or ‘Slave-Drivers’) are all oddballs. It’s not the ‘downside’ of Parkinson’s but quite the reverse! No matter if, we are the detained or detainee, it’s Parkinson’s that rules the roost over both the afflicted and affected. It’s baffling, befuddling, ungainly and just plain confounding. So demanding is Parkinson’s, that it so often leaves us depleted of any semblance of control, there’s not much else to do but LAUGH! Laugh so hard, we can control out tears (we call it schlapp-lakking– great word!). My sister and me would so often be overcome by schlapp-lakking always in the most exhausting circumstances. I’m one and a half years her junior, and was helping her move from her house (she’d raised her kids in…and she was a HOARDER and a half!), in a hurry. I’m a stocky pit-bull. She’s a stick insect with a seriously over-blown sense of her physical acumen. CAVEAT - this is just my humour; I even dug a hole for her cat (very much alive then). It sounds wrong, but the British are so obsessively compulsive about animals. Vetting potential humans for re-homing involves criminal background, sodium pentothal, lie detector tests, and signing your life away; because those in control will visit said adoptee and give it stuffed, fluffy rodents to play with while under ‘observation’. However, this mog was running out of time – no takers, and she was moving to a flat (apartment) which does not take animals - few do, and you have to make a pact with Beelzebub and sign your kidneys away. Thus, it was reasonable to assume, at the 11th hour, Fluffy was not going to make it. He did…just, and I had to fill the bloody hole in. I digress, sorry! Last couple of days, and with the meter ticking on exchange, she wanted to take a ‘couple’ of wee plants. I groaned noisily, but can never refuse her anything. So we end up schlepping several TREES in tubs probably weighing in at 300+lb. per – UP the steep garden to the waiting car…that’s when schlepping sets in – fits of sidebusting giggles turn into uncontrollable roaring – followed by a quick bathroom break. Ladies who have born children will understand that heavy lifting whilst laughing hysterically will lead to ‘accidents’! A job that would’ve taken a couple of blokes 10 minutes, took us half a day, but worth every moment! continued from page 3 Low-Level Pesticides… Dr. Joseph Mercola producing cells within your brain, known as the substantia nigra, required for normal movement, begin to die. As the disease progresses, symptoms include increasingly slow motor function, tremors when at rest, muscle rigidity, and abnormalities in your walk. Up to one million Americans currently have Parkinson’s and 60,000 more are diagnosed each year. As a disease that currently cannot be cured, prevention of Parkinson’s is crucial. People over 60 years of age used to make up the majority of those afflicted, and with increasing lifestyle factor risks like chronic exposure to pesticides and a generally increasing toxic burden, cases are now being reported regularly in people under 50 and even under 40 years of age. Parkinson’s is strongly related to lifestyle choices, which is good news because it means there are steps you can take to modify your risk. Eating a varied whole-foods diet that includes healthful veggies like peppers appears to be one simple way to lower your risk, especially since a lack of folate is also linked to Parkinson’s (and veggies are the only source of this important vitamin; most vitamins contain the semi-synthetic analog known as folic acid). Additionally, the following nutrients are also important and may help you to significantly lower your risk: Vitamin D: There is a correlation between insufficient levels of vitamin D and the development of early Parkinson’s disease. It used to be thought that vitamin D deficiency is a symptom of Parkinson’s, but recent research squarely implicated vitamin D deficiency as one of the causes of Parkinson’s. The best way to optimize your vitamin D level is through appropriate midday sun exposure or a safe tanning bed, as optimizing your body’s own production virtually eliminates any risk of overdose. Animal-based omega-3 fats: These may protect against Parkinson’s by preventing the misfolding of a protein associated with a gene mutation in neurodegenerative diseases like Parkinson’s and Huntington’s. Animalbased omega-3 fats contain two fatty acids crucial to human health, DHA and EPA. Most of the neurological benefits of omega-3 oils are derived from the DHA, which is one of the major building blocks of your brain. Adding omega-3 fats to your diet, via wild-caught fish or a supplement like krill oil, is important. Coenzyme Q10 (CoQ10): One study found that the progression of Parkinson’s disease was significantly slower in people taking the highest dose of CoQ10. If you’re taking statin cholesterol-lowering drugs, this issue is particularly important as these medications deplete your body of CoQ10, making supplementation with CoQ10 (or ideally, the reduced form, called ubiquinol) important. What was I saying? Ah, yes…something about Parkinson’s! Over the coming months, I intend to offer you my ‘take’ on living with PD. That’s because I have a 6’3’’ 230lb festinating bloke who I happen to be the caregiver for. He’s Shaky Pete - Puff Daddy – Butterball – my husband Pete who is in his 16th year of PD. Green Tea: Green tea’s polyphenols offer neuroprotection that might benefit Parkinson’s patients. Caffeine, which is doparminergic (stimulates dopamine release), may be one more reason why green tea has also been linked to a lower Parkinson’s risk. In fact, the daily caffeine equivalent in two to four cups of coffee may modestly improve Parkinson’s movement symptoms as well. vvvvv Finally, your best bet for minimizing health risks from pesticide exposure is to avoid them by eating organic as much as possible and investing in a good water filtration system for your home or apartment. If you know you have been exposed to pesticides, the lactic acid bacteria formed during the fermentation of kimchi may also help your body break down pesticides. By including fermented foods, like kimchi, in your diet, is a wise strategy to help detox the pesticides that enter your body. Lisa Vanderburg is the European International Research Specialist and Staff Writer for Friends of Parkinson’s. Wishing You Health and Happiness From Across The Pond! Lisa Venderburg Page 6 • October 2015 • Friends of Parkinson’s vvvvv Dr. Mercola authored two New York Times best sellers, The No-Grain Diet and The Great Bird Flu Hoax. An osteopathic physician (DO), his primary passion is transforming the conventional medical paradigm in the United States. Visit www.DrMercola.com, Mercola.com. Events & Resources October TAI CHI FOR BALANCE, Mondays & Wednesdays, 11am. West Flamingo Active Adult Cntr. 6255 W, Flamingo. 702-455-7742. MOVE I EXERCISE CLASSES, Tuesdays & Thursdays, 11:15am to 12:15pm, (seated exercise for anyone with neurodengerative disorders) Lou Ruvo Center for Brain Health. 888 W. Bonneville, call Lakita Arrington. 702-483-6032. MOVE II EXERCISE CLASSES, Tuesdays & Thursdays, 12:30pm to 1:30pm, (standing exercise for anyone with neurodengerative disorders) Lou Ruvo Center for Brain Health. 888 W. Bonneville, contact Lakita Arrington. 702-483-6032. ($10 drop in price). ZUMBA GOLD, Wednesdays, 9-10am. 2651 Paseo Verde Pkwy, Suite 180. Contact Kim Riddle, 702616-4902. ($5/per session or 5 sessions for $20). AGELESS WOMAN WORKOUT, Tuesdays & Thursdays, 9-10am. 2651 Paseo Verde Pkwy, Suite 180. Contact Kim Riddle, 702-616-4902. ($5/per session or 5 sessions for $20). GENTLE YOGA, Tuesdays & Thursdays, 10-11am. 2651 Paseo Verde Pkwy, Suite 180. Contact Kim Riddle, 702-616-4902. ($5/per session or 5 sessions for $20). AEROBIC FUN EXERCISE CLASSES, Mondays, Wednesdays & Fridays, 9:30am. Fern Adair Conservatory of the Arts, 3265 E. Patrick Lane. TAI CHI FOR BALANCE, Fridays, 9-10am. Desert Breeze Community Center, 8275 Spring Mountain Road, LV. 702-455-8334. Facilitator Tamalyn Taylor MS. Dance for Parkinson's, Wednesdays, 12:301:30pm, Las Ventanas (open to the public). YOUNG ONSET PD SUPPORT GROUP of Southern Nevada, Cidney Donahoo, 702-326-6831. [email protected]. DBS SUPPORT GROUP of Southern Nevada, Kip Smith, 702-755-5704. [email protected]. LET'S MEET & EAT FOR CAREGIVERS, • 2nd Friday, 10am, H2U Mountain View Office, 3150 N. Tenaya Way, Suite 114. Call 702-3814141 for info. • 3rd Tuesday, 2pm,Vida Training Center, 1785 E. Sahara Ave., Suite 430. Call 702-381-4141. Veteran'S INVOLVED PARKINSON'S (VIP), 1pm (1st Tuesday) Friends of Parkinson's Office, 2400 N. Tenaya Way. 702-381-4141. PWP BRUNCH BUNCH, • 1st Saturday, 9am. Mountain View Hospital, 3100 N. Tenaya Way, First floor. 702-381-4141. PWP BRUNCH BUNCH, 4th Tuesday, 1:30pm. H2U Mountain View Office, 3150 North Tenaya Way, Suite 114. 702381-4141. HEALTHSOUTH DESERT CANYON HOSPITAL, The Parkinson’s Disease Rehabilitation Program provides a full range of rehabilitative services aimed at meeting the global range of medical, psychological and functional needs of Parkinson’s patients. 9175 W. Oquendo Rd., Las Vegas. For additional information please call 702-252-7342. SPECIAL ANNOUNCEMENT AbbVie Usa Llc Speak at each Friends of Parkinson's Support Groups call 702-381-4141 to PPPPPPP Saturday March 26, 2016 FUNNY BUNNY RACE ONLINE Early sign ups @ www.Active.com Make this day special and then pay-it-forward! Location: The Elks Lodge, 4100 West Charleston Blvd, LV Friends of Parkinson’s • October 2015 • Page 7 Page 8 • October 2015 • Friends of Parkinson’s